Tuesday, January 10, 2012

Ford promising 12,000 jobs

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Ford promising 12,000 jobs ??



?????Tuesday 10th January, 2012??Source: News & Observer ??
William Clay Ford Jr., right, and Alan Mulally, Ford executive chairman and CEO respectively, show off the 2013 Ford Fusion Hybrid Energi Plug-in at the 2012 North American International Auto Show in Detroit.

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Tuesday 10th January, 2012


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Monday, January 9, 2012

Nigerian sect kills 15; Christians vow defense

Bodies of people alleged to have been killed in a Friday attack on a town hall meeting of the Christian Igbo ethnic group lie on the floor in a hospital morgue in Mubi, in the Adamawa state of northern Nigeria, Saturday, Jan. 7, 2012. The town hall attack, which left at least 20 dead, is one of a string of deadly attacks claimed by radical Muslim sect Boko Haram, which has promised to kill Christians living in Nigeria's largely Muslim north. At least 44 people have been killed in the last few days alone. (AP Photo)

Bodies of people alleged to have been killed in a Friday attack on a town hall meeting of the Christian Igbo ethnic group lie on the floor in a hospital morgue in Mubi, in the Adamawa state of northern Nigeria, Saturday, Jan. 7, 2012. The town hall attack, which left at least 20 dead, is one of a string of deadly attacks claimed by radical Muslim sect Boko Haram, which has promised to kill Christians living in Nigeria's largely Muslim north. At least 44 people have been killed in the last few days alone. (AP Photo)

MAIDUGURI, Nigeria (AP) ? A radical Muslim sect attacked a church worship service in Nigeria's northeast during assaults that killed at least 15 people, authorities said Saturday, as Christians vowed to defend themselves from the group's widening sectarian fight against the country's government.

The attacks by the sect known as Boko Haram came after it promised to kill Christians living in Nigeria's largely Muslim north, exploiting long-standing religious and ethnic tensions in the nation of more than 160 million people. The pledge by the leader of an umbrella organization called the Christian Association of Nigeria now raises the possibility of retaliatory violence.

In the last few days alone, Boko Haram has killed at least 44 people, despite the oil-rich nation's president declaring a state of emergency in regions hit by the sect.

Speaking Saturday to journalists, Pastor Ayo Oritsejafor, president of the Christian Association of Nigeria, vowed the group's members would adequately protect themselves from the sect. He declined to offer specifics, raising concerns about retaliation.

"We have decided to work out means to defend ourselves against these senseless killings," Oritsejafor said.

He later added: "We cannot sit back and watch people being slaughtered like animals everyday, going to the church, shooting people, killing them. This is unacceptable."

In Yola, the capital of Adamawa state, gunmen covered their faces with black cloth when they attacked Apostolic Church on Friday night, local police commissioner Ade Shinaba said. Shinaba said at least eight worshippers died in that attack.

At a nearby beauty salon, at least three others were killed in a similar attack.

"Three gunmen with their faces covered with black cloth burst into my salon and started shooting at customers, chanting, 'God is great, God is great,'" said Stephen Tizhe, 35.

Responding to the violence, Adamawa state Gov. Murtala Nyako ordered a 24-hour curfew throughout the rural state. The violence comes ahead of a planned gubernatorial election later this month.

In the town of Potiskum in Yobe state, gunmen set two banks ablaze with gasoline bombs, starting a gunfight with police that lasted three hours Friday, local police commissioner Tanko Lawan said. At least two people were killed in the fight, he said.

On Saturday, sect gunmen also shot and killed two Christian students who attend the University of Maiduguri in nearby Borno state, local police commissioner Simeon Midenda said.

No arrests have been made in any of the attacks, authorities said.

The attacks came after gunmen claimed by Boko Haram attacked a town hall earlier Friday in Mubi, Adamawa state, killing at least 20 people who had gathered for a meeting of the Christian Igbo ethnic group. On Thursday night, the sect also attacked a church in Gombe state, killing at least eight people.

Boko Haram, whose name means "Western education is sacrilege" in the local Hausa language, is responsible for at least 510 killings last year alone, according to an Associated Press count. It has targeted churches in the past in its campaign to implement strict Shariah law across Nigeria.

The group claimed responsibility for attacks that killed at least 42 people in Christmas Day strikes that included the bombing of a Catholic church near Abuja. The group also claimed an August suicide car bombing that targeted the U.N. headquarters in the capital, killing 25 people and wounding more than 100.

Nigeria's central government has been slow to respond to the sect. On Dec. 31, President Goodluck Jonathan declared regions of Borno, Niger, Plateau and Yobe states to be under a state of emergency, meaning authorities can make arrests without proof and conduct searches without warrants. He also ordered international borders near Borno and Yobe state to be closed.

However, the areas where the recent church and town hall attacks happened are not in the areas marked by the president.

Boko Haram promised to begin attacking Christians in Nigeria's north several days before the recent violence. The new killings have sparked fears among Christians living in the north about the group and caused some to flee. There also has been at least one report of retaliatory violence against Muslims living in Nigeria's mostly Christian south in recent days as well.

___

Associated Press writer Lekan Oyekanmi in Abuja, Nigeria contributed to this report.

___

Jon Gambrell reported from Lagos, Nigeria and can be reached at www.twitter.com/jongambrellAP.

Associated Press

Source: http://hosted2.ap.org/APDEFAULT/cae69a7523db45408eeb2b3a98c0c9c5/Article_2012-01-07-AF-Nigeria-Violence/id-95709d1ad5c14647a496216ec895278a

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Sunday, January 8, 2012

nbcchicago: RT @bjlutz: Cardinal George apologizes for linking gay parade with KKK activism http://t.co/EzjmKePr

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Lawmaker targets TSA's big chunk of change

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Travelers left $376,480.39 in loose change in the bottoms of plastic bins at airport security checkpoints in the 2010 fiscal year, according to the Transportation Secuirty Administration.

By Harriet Baskas, msnbc.com contributor

Hate hidden travel fees? Then pay attention when collecting your belongings at the airport security checkpoint.?

In fiscal year 2010, travelers left $376,480.39 in loose change in the bottoms of plastic bins at the checkpoints, according to the Transportation Security Administration.?In 2009, the abandoned coins totaled more than $399,000. ?

?Passengers say their six pennies don?t matter,? said TSA spokesperson Nico Melendez. ?But it adds up.?

Live Poll

Where should unclaimed change from airport security checkpoints go?

Melendez said all the unclaimed pennies, nickels, dimes and quarters are turned over to the TSA finance office. After being documented and counted, the money ends up in the coffers of the TSA, which is authorized by law to spend that money as it sees fit.

But Rep. Jeff Miller (R-Fla.) wants to change what happens to that big chunk of change.

Miller?has introduced legislation that?would direct the TSA to transfer unclaimed money recovered at airport security checkpoints to the United Service Organizations (USO), a private nonprofit that operates centers for the military at 41 U.S. airports.

Miller first introduced the bill in 2009, but it didn?t get much traction. Now that he?s the chairman of the House Committee on Veterans' Affairs, he?s trying to move the bill forward again.?

?The money left behind at the airport checkpoints belongs to taxpayers,? said Miller spokesperson Dan McFaul. ?The congressman feels giving it to the USO to help with onsite airport service for active members of the military would be a good use for it.?

McFaul said the bill is currently being considered by two committees ? Homeland Security and Transportation ? and that ?the immediate focus is to get a hearing and get support.?

The USO, which did not initiate the campaign to redirect unclaimed checkpoint change, is nonetheless honored by Miller?s idea.

?Absolutely,? said?Frank Thorp,?USO's senior vice president for marketing and?communications. ?Any dollar amount we get from the American people goes toward the troops and families who need us most. Our centers provide a warm and comforting place where troops can connect with family via Internet or telephone, play a video game, catch a movie, have a snack or just put their feet up and relax.? ?

As a federal agency, the TSA has no official position on the pending legislation, but Melendez says: ?If people don?t want the TSA to get that money, they can do what I do. If I have spare change in my pocket, I put it in my briefcase so I don?t leave it behind.?

More on Overhead Bin

Find more by Harriet Baskas on?Stuck at The Airport.com?and follow her on?Twitter.

Source: http://overheadbin.msnbc.msn.com/_news/2012/01/06/9979819-lawmaker-targets-tsas-big-chunk-of-change

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Friday, January 6, 2012

Scientists crack medieval bone code

Tuesday, January 3, 2012

Two teams of Michigan State University researchers ? one working at a medieval burial site in Albania, the other at a DNA lab in East Lansing ? have shown how modern science can unlock the mysteries of the past.

The scientists are the first to confirm the existence of brucellosis, an infectious disease still prevalent today, in ancient skeletal remains.

The findings, which appear in the American Journal of Physical Anthropology, suggest brucellosis has been endemic to Albania since at least the Middle Ages.

Although rare in the United States, brucellosis remains a major problem in the Mediterranean region and other parts of the world. Characterized by chronic respiratory illness and fever, brucellosis is acquired by eating infected meat or unpasteurized dairy products or by coming into contact with animals carrying the brucella bacteria.

Todd Fenton, associate professor of anthropology, said advanced DNA testing at MSU allowed the researchers to confirm the existence of the disease in skeletons that were about 1,000 years old.

"For years, we had to hypothesize the cause of pathological conditions like this," Fenton said. "So the era of DNA testing and the contributions that DNA can make to my work are really exciting."

Here's how the discovery came about.

Fenton and a group of MSU graduate students were serving as the bioarcheologists, or bone specialists, for a multinational team of archaeologists excavating sites in the ancient Albanian city of Butrint. Once a large Roman colony, Butrint in its final centuries served as an outpost of the Byzantine Empire until it was abandoned in the Middle Ages due to flooding.

Fenton and his team developed biological profiles of the human remains, which included determining sex, age and skeletal pathologies, or health histories. Vertebrae from two of the Byzantine-era skeletons ? both adolescent males from the 10th century to the 13th century ? had significant lesions, leading the researchers to theorize the boys had suffered from tuberculosis.

Samples of the ancient bone were sent to the forensic DNA lab in East Lansing, which is headed by David Foran, director of MSU's Forensic Science Program. Foran and his team of graduate students took tiny portions of the bone, extracted DNA and tested it for any residual DNA that might still exist from the expected pathogen.

But the results came back negative for tuberculosis.

Fenton's team re-examined the bones that tested negative for tuberculosis and concluded the disease might instead be brucellosis. The infection from brucellosis and tuberculosis causes similar damage ? basically eating away the bone ? although no one had ever confirmed brucellosis in human bone recovered from an archaeological site.

Foran's team then developed a different set of tests for detecting the brucella bacteria and undertook a new round of testing on the diseased vertebrae.

This time the results came back positive for brucellosis.

Foran said the collaboration on the project highlights the benefits of modern science and interdisciplinary research, even when the respective research teams are some 5,000 miles apart.

"In this case it was a combination of inquisitiveness, persistence and of course collaboration," Foran said. "It is amazing to find something brand new in something that is a thousand years old."

###

Michigan State University: http://www.newsroom.msu.edu

Thanks to Michigan State University for this article.

This press release was posted to serve as a topic for discussion. Please comment below. We try our best to only post press releases that are associated with peer reviewed scientific literature. Critical discussions of the research are appreciated. If you need help finding a link to the original article, please contact us on twitter or via e-mail.

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Source: http://www.labspaces.net/116378/Scientists_crack_medieval_bone_code_

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Thursday, January 5, 2012

Benefits of statin therapy may extend beyond lowering lipids

ScienceDaily (Jan. 4, 2012) ? People with high cholesterol are at risk of heart attack and stroke because atherosclerotic plaques within their arteries can rupture triggering the formation of a blood clot called an occlusive thrombus that cuts off the blood supply to their heart or brain.

For years, scientists have studied the cause of this abnormal clotting. Now, a study led by researchers from the University of North Carolina at Chapel Hill School of Medicine has identified a molecular pathway that leads to this abnormal blood clotting. The researchers then turned off the pathway by using a popular class of cholesterol-lowering drugs, statins.

The research was performed using humans, monkeys and mice with highly elevated blood lipid levels. It indicated that elevated levels of oxidized low density lipoprotein (LDL) induces a molecule called "tissue factor" that triggers clotting. The study appears online in the January 3, 2012 issue of the Journal of Clinical Investigation.

"Statins have been shown to have antithrombotic activity in several previous studies. However, I believe our study is the first to elucidate how statins reduce the activation of the blood clotting process independently of their lipid lowering activity, said senior study author Nigel Mackman, PhD, FAHA. Mackman is the John C. Parker Distinguished Professor of Hematology in the Department of Medicine and Director of the UNC McAllister Heart Institute.

Additionally, Mackman noted that statins "only target the 'bad and inducible tissue factor', not the good one used in normal clotting, and therefore should not be associated with the increased bleeding risk that is a typical side effect of anticoagulant drugs currently on the market."

Mackman has spent the last twenty-five years studying tissue factor, the number one initiator of clotting in the body. Tissue factor normally resides outside the blood vessels, only coming into contact with blood after an injury, such as cutting your finger. However, it is expressed at high levels under certain abnormal conditions, such as inside atherosclerotic plaques, and gets turned on in a special subset of blood cells called monocytes. Mackman wondered if this was the cause for the abnormal clotting seen in patients with high cholesterol.

To test his hypothesis, Mackman and his colleagues analyzed humans, monkeys and mice with high cholesterol. They found that all three groups have elevated levels of tissue factor in the circulation.

Then the researchers treated the mice and monkeys with simvastatin, a drug widely used to treat high blood cholesterol levels. They showed that simvastatin reduced levels of oxidized low density lipoprotein and circulating tissue factor which normalized coagulation without altering plasma cholesterol levels. These results suggest that oxidized low density lipoproteins induce tissue factor expression on monocytes and this contributes to formation of an occlusive thrombus after plaque rupture.

"Though statin therapy is primarily prescribed to lower cholesterol, some added benefits are its anti-inflammatory and antithrombotic activities," said Mackman. "In terms of drug development, I think we should be trying to better understand the antithrombotic activities of statins so we can develop safer antithrombotic drugs that target the expression of inducible tissue factor."

The research was funded largely by the National Heart, Lung and Blood Institute, a component of the National Institutes of Health. The majority of this work was performed by A. Phillip Owens III, PhD. Other study co-authors from Dr. Mackman's lab at UNC were Silvio Antoniak, PhD; Julie C. Williams; and Jianguo Wang, PhD.

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The above story is reprinted from materials provided by University of North Carolina School of Medicine.

Note: Materials may be edited for content and length. For further information, please contact the source cited above.


Journal Reference:

  1. A. Phillip Owens, Freda H. Passam, Silvio Antoniak, Stephanie M. Marshall, Allison L. McDaniel, Lawrence Rudel, Julie C. Williams, Brian K. Hubbard, Julie-Ann Dutton, Jianguo Wang, Peter S. Tobias, Linda K. Curtiss, Alan Daugherty, Daniel Kirchhofer, James P. Luyendyk, Patrick M. Moriarty, Shanmugam Nagarajan, Barbara C. Furie, Bruce Furie, Douglas G. Johns, Ryan E. Temel, Nigel Mackman. Monocyte tissue factor?dependent activation of coagulation in hypercholesterolemic mice and monkeys is inhibited by simvastatin. Journal of Clinical Investigation, 2012; DOI: 10.1172/JCI58969

Note: If no author is given, the source is cited instead.

Disclaimer: This article is not intended to provide medical advice, diagnosis or treatment. Views expressed here do not necessarily reflect those of ScienceDaily or its staff.

Source: http://www.sciencedaily.com/releases/2012/01/120104153743.htm

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Sunday, January 1, 2012

Casio?s New G-SHOCK Connects To Smartphones, Shows Incoming Calls, Emails, SMS

g-shockCasio Japan is planning [JP] to roll out the G-SHOCK GB-6900 on March 16 next year, a wristwatch that connects to certain smartphones via Bluetooth LE (LE=low-energy, a standard that's baked into Bluetooth 4.0). The device will be compatible with the Medias LTE N-04D Android phone from NEC (to be released next year) and NEC's Medias PP N-01D. Casio says that the G-SHOCK not only synchronizes the time with the phones but also shows incoming calls, emails, or SMS on its display. Users can also switch their handsets to vibration mode by pushing a button on the watch or set alarms.

Source: http://feedproxy.google.com/~r/Techcrunch/~3/lSn26Bw9D0w/

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